The CLCN4 gene encodes the CLC-4 protein, a Cl-/H+ exchanger expressed primarily in brain and skeletal muscle. The CLC-4 protein is expressed in intracellular organelles and is thought to be involved in ion homeostasis of endosomes and intracellular trafficking. The CLCN4 gene is located on the X-chromosome and mutations in this gene lead to frameshift, missense and splice site variants, and chromosomal microdeletions in the CLC-4 protein. Dysfunction of this protein underlies the clinical phenotype of CLCN4-related conditions, symptoms of which include intellectual disability and other problems such as epilepsy, behavioural disorders, movement disorders and microcephaly.
CLCN4-related conditions are extremely rare, so far just over 120 individuals worldwide with a CLCN4 gene change have been identified. However, symptoms vary widely in their extent and severity and as genetic testing becomes more widely available, more individuals with undiagnosed intellectual disability are likely to be diagnosed with CLCN4-related conditions.
More research is desperately needed into CLCN4 and the protein, CLC-4. We are proud to support the Cure CLCN4 Annual Scientific Meeting to further the understanding of the basic biology of CIC-4 and investigate therapeutic options for these patients.
We provide a range of electrophysiology platforms to study ion channels and transporters in native and heterologous cells, intracellular organelles and membranes:
These platforms can be used to study basic mechanisms of CLC-4 function and could be used to investigate variants of unknown significance of CLCN4-related conditions, as well as screening for potential new drugs.
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